Non-alcoholic fatty liver disease is a condition in which the liver accumulates fat without excessive alcohol consumption.
Abdominal obesity is a major risk factor for Non-alcoholic fatty liver disease development and progression. Belly obesity causes liver diseases like NAFLD:
Insulin resistance, which reduces cell sensitivity, sometimes occurs with obesity. Insulin resistance raises blood insulin levels. High insulin levels cause hepatic fat buildup.
Free fatty acids enter the bloodstream from belly fat. These fatty acids are absorbed by the liver, causing liver cell fat.
Inflammatory cytokines and adipokines are released by belly fat, especially visceral fat. These drugs can cause liver inflammation, which accelerates Non-alcoholic fatty liver disease.
When adipose tissue, especially abdominal fat, malfunctions, pro-inflammatory chemicals are released. Adipose tissue malfunction causes hepatic and systemic inflammation.
The liver stores excess blood fat, especially from abdominal fat storage, as triglycerides. Lipid buildup in the liver can cause Non-alcoholic fatty liver disease.
Patients with liver health issues or Non-alcoholic fatty liver disease should consult with hepatologists, dietitians, and other specialists to create a customized treatment strategy.
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